My system would be based on a point system. As a person commits crimes they would accumulate points when convicted. Each conviction would have a point value attached. These points would start at day 1 of life.
There would be a defined number of high security prison beds assigned based on the population. Points would accumulate for all crimes but the felon would have to commit violent crimes to be eligible for the death penalty. The people with the most points would be bumped off the end.
So people who commit crimes in prison could also run the risk of execution. Also it is only likely that the worst criminals would be executed regardless of race or wealth.
The death penalty should be federal only. So points would be compared to all felons across the nation.
Finally there should be no such thing as attempted murder. If a person attempts to kill someone and is convicted of it the victims good fortune shouldn't benefit the felon. They should be charged as if they we successful in killing the victim.
Monday, January 7, 2008
When we are going to die?
There is an answer or method to determine your life expectancy based on the telomere for example.
Telomeres are unique protein–DNA structures. They are effectively a life line for cells. They determine how many times a cell can split.
Look at it this way each copy of something gets progressively degraded from the original. If you were to live forever the person you would become would be an absolute physically altered genetic mess assuming current conditions. Its a hard concept but if we were to make comparisons between peoples telomeres with sample from a wide range of cells we could determine their life expectancy assuming no other factors take(kill) them sooner.
Also check out my website. There is a link to a New kind of science that will show you that no matter the situation there is a predetermined out come. Chaos theory is just bad math so don't even try it. My website is: http://thinkbiggar.blogspot.com/"
A pivotal finding in the understanding of somatic cell bi-ology was the observation that normal somatic cells have a finite replicative life span (3). That is, they are capable of a finite number of cell divisions, after which they under go what has been termed replicative senescence and are inca-pable of further cell division. The mechanism underlying the replicative clock that monitors this process has evoked considerable attention, and it is in this context that telo-mere function has been of particularly intense interest.
The most widely accepted paradigm relating telomere function to cellular aging and replicative senescence is based on the observation that in normal somatic cells telomeres shorten with each cell division (4, 5). This telomere shortening has been attributed to the primer requirement for DNA syn- thesis during chromosomal replication, and results in in- complete replication and a loss of terminal telomeric repeats with each cell division (6). Telomeres thus shorten progressively with successive cell divisions, and telomere length in a somatic cell may thus reflect the replicative his-tory of that cellular lineage. "
Can we reverse old age?
http://tasciences.com/?gclid=CPW614f75ZACFQFjHgodcBUmOA
Source(s):
http://www.jem.org/cgi/reprint/190/2/153.pdf
http://thinkbiggar.blogspot.com/
Telomeres are unique protein–DNA structures. They are effectively a life line for cells. They determine how many times a cell can split.
Look at it this way each copy of something gets progressively degraded from the original. If you were to live forever the person you would become would be an absolute physically altered genetic mess assuming current conditions. Its a hard concept but if we were to make comparisons between peoples telomeres with sample from a wide range of cells we could determine their life expectancy assuming no other factors take(kill) them sooner.
Also check out my website. There is a link to a New kind of science that will show you that no matter the situation there is a predetermined out come. Chaos theory is just bad math so don't even try it. My website is: http://thinkbiggar.blogspot.com/"
A pivotal finding in the understanding of somatic cell bi-ology was the observation that normal somatic cells have a finite replicative life span (3). That is, they are capable of a finite number of cell divisions, after which they under go what has been termed replicative senescence and are inca-pable of further cell division. The mechanism underlying the replicative clock that monitors this process has evoked considerable attention, and it is in this context that telo-mere function has been of particularly intense interest.
The most widely accepted paradigm relating telomere function to cellular aging and replicative senescence is based on the observation that in normal somatic cells telomeres shorten with each cell division (4, 5). This telomere shortening has been attributed to the primer requirement for DNA syn- thesis during chromosomal replication, and results in in- complete replication and a loss of terminal telomeric repeats with each cell division (6). Telomeres thus shorten progressively with successive cell divisions, and telomere length in a somatic cell may thus reflect the replicative his-tory of that cellular lineage. "
Can we reverse old age?
http://tasciences.com/?gclid=CPW614f75ZACFQFjHgodcBUmOA
Source(s):
http://www.jem.org/cgi/reprint/190/2/153.pdf
http://thinkbiggar.blogspot.com/
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